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Normal values have been established for distal latencies from the various sites of stimulation on various mixed nerves to the appropriate muscles. When one stimulates the median nerve at the wrist, for example (see electrode 1 and segment A in Fig. 45-3), the latency for motor conduction through the carpal tunnel to the median-innervated thenar muscles is always less than 4.5 ms in healthy adults. Similar normal values have been compiled for orthodromic and antidromic sensory conduction velocities and distal latencies (see Table 45-1) in all the main peripheral nerves. Disease processes that preferentially injure the fastest-conducting, large-diameter bers in peripheral nerves reduce the maximal conduction velocity because the remaining thinner bers conduct more slowly. In most neuropathies, all of the axons are affected either by a fairly uniform dying-back phenomenon or by wallerian degeneration as described in Chap. 46, and nerve conduction velocities are then less informative. This is true for typical alcoholic-nutritional, carcinomatous, uremic, diabetic, and other metabolic neuropathies, in which conduction velocities range from low normal to mildly slowed. In these so-called axonal neuropathies, the motor and sensory nerve amplitudes are diminished (see below). By contrast, demyelinating neuropathies of the acute (Guillain-Barre, diphtheria) and chronic types such as chronic in am matory, metachromatic leukodystrophy, Krabbe disease, and the common type of Charcot-Marie-Tooth disease show marked slowing of conduction and, in the case of the acquired demyelinating diseases, there is also dispersion of the action potential and conduction block (see later).

3. Using the results from Steps 1 and 2 show that z = 1 e2 tan p (2.75)

We must require that 2a 3. If we do not impose that restriction, we allow for the possibility of division by 0 in the last step.

Fig. 12-16

There are several variations on butter y spreads traded today. Butter y spreads vary slightly in design from equity options trading, interest rate trading, and commodity option trading. We focus on a couple of common option variations of the butter y. Butter y spreads are not speci cally neutral delta spreads, but they are frequently very close to neutral delta with a very small long or short delta depending on the spread.

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individual is exposed, particularly in the far higher northern and southern latitudes This phenomenon of molecular mimicry (a shared antigen between the virus and CNS myelin, the oligodendrocyte, or cerebral vessels) has been invoked as a mechanism in several diseases, notably in rheumatic fever, certain paraneoplastic diseases, and Guillain-Barre syndrome, but remains speculative in MS The role of humoral and cellular factors in the production of MS plaques is not fully understood The deposition of immunoglobulin in the plaques of patients with acute and relapsing-remitting disease, but not those with progressive MS, has been alluded to earlier That the humoral immune system is involved is evident from the presence in the CSF of most patients of oligoclonal immune protein antibodies, which are produced by B lymphocytes within the CNS.

To install your command-line application on your Android server, you need to use adb. adb is your connection to the Android server, which is started with your Emulator. adb contains many useful functions that allow you to interact with your Android server; one of these enables you to install applications. Table 6-1 lists and describes the commands that adb accepts.

Sera from patients with MS (and some normal controls), when added to cultures of nervous system tissue from newborn mice in the presence of complement, can damage myelin, inhibit remyelination, and block axonal conduction Antibodies to oligodendrocytes are present in the serum of up to 90 percent of patients in some studies but far less frequently in others Of note also are the ndings of Berger and colleagues that show a relationship between the clinical progression of early and monosymptomatic MS and the presence of serum autoantibodies against oligodendrocyte glycoprotein and myelin basic protein They have reported that the absence of antibodies, a nding in 38 percent of their patients, identi ed a group with more benign disease, at least for several years Whether this further indicates, as they suggest, that humoral immune mechanisms are essential to the production of MS is uncertain.

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